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Intestinal Perforation Secondary to Bortezomib-Induced Autonomic Neuropathy

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dc.contributor.author Sánchez-Salas, José-Antonio
dc.contributor.author Moreno-Belmonte, María-José
dc.contributor.author Poveda-García, Andrea
dc.contributor.author Ruiz-Ruiz, Estela
dc.contributor.author Soler-Espejo, Eva
dc.contributor.author Cabanas-Perianes, Valentín
dc.contributor.author García-Hernández, Ana-María
dc.date.accessioned 2026-03-06T14:04:35Z
dc.date.available 2026-03-06T14:04:35Z
dc.date.issued 2025-04
dc.identifier.citation Sánchez Salas JA, Moreno Belmonte MJ, Poveda García A, Ruiz Ruiz E, Soler Espejo E, Cabanas Perianes V, et al. Intestinal Perforation Secondary to Bortezomib-Induced Autonomic Neuropathy. Clinical Case Reports. abril de 2025;13(4):e70340. doi:10.1002/ccr3.70340
dc.identifier.issn 2050-0904
dc.identifier.uri https://sms.carm.es/ricsmur/handle/123456789/24588
dc.description.abstract It is essential to evaluate both the patient's prior conditions and the severity of the current clinical presentation when deciding on BTZ toxicity management. It seems prudent to consider the permanent discontinuation of the drug in patients who have experienced at least grade 3 intestinal neuropathy and have structural abnormalities or other risk factors for intestinal perforation. Trial Registration: NCT03710603.
dc.language.iso eng
dc.publisher WILEY
dc.rights Atribución/Reconocimiento 4.0 Internacional
dc.rights.uri https://creativecommons.org/licenses/by/4.0/deed.es
dc.title Intestinal Perforation Secondary to Bortezomib-Induced Autonomic Neuropathy
dc.type info:eu-repo/semantics/article
dc.identifier.pmid 40171013
dc.relation.publisherversion https://onlinelibrary.wiley.com/doi/10.1002/ccr3.70340
dc.type.version info:eu-repo/semantics/publishedVersion
dc.identifier.doi 10.1002/ccr3.70340
dc.journal.title Clinical Case Reports


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